Monday, April 2, 2012

PHYSICS IS FUN: Tennis Anyone?

So today I want to discuss an important concept in ultrasound and it involves physics.  Yes, I know, you thought that once you took the MCAT and got into medical school, you would be done with physics, but  no!  To truly understand ultrasound, you gotta know a little physics.

Lets start with how different tissue types create different images on the screen.  The sound waves sent out from the probe strike the body and return to the probe.  Depending on what they strike depends on how much or how little of the initial sound beam return.

Lets image that we have a bucket of tennis balls and we are ECC.


If we thrown ten tennis balls at the wall, how many come back to us?


All of them, correct?  This is due to the fact that the wall is dense, just like a gallstone or bone is dense.  In US, those sound waves strike this dense "wall" and cannot penetrate further, just like the tennis balls cannot go through the wall.  Since the solid object blocks the sound waves from penetrating further into the body, there is a void of sound waves posterior to the solid.  This void of sound is called Acoustic Shadowing.




This is a useful property as it can help us determine pathologic states or creates landmarks for us to follow.

So what if we throw our tennis balls at the curtain between resus beds 3 and 4, how many will come back to us?



It depends on the stiffness of the curtain, of course, but a certain amount will continue through the curtain and some will bounce back to us.  This is what occur in soft tissues.  Some sound waves bounce back to the machine and some penetrate through to hit structures underneath.  As with the curtain, the strength to which the tennis balls bounce back varies with the stiffness or density of the curtain.  Same for ultrasound: the amplitude of the returning sound wave varies depending on the density of the soft tissue it strikes.  As the sound waves go through each progressive layer of soft tissue, the amplitude becomes weaker and weaker, until finally there is not any sound left.


This is how we can see different tissue planes on ultrasound and determine the difference between different tissue types.



Now let's image that we take our bucket of tennis balls and throw them down the aisle toward the fishbowl.  How many will return to us?


None, right?  They all go until they hit the bowl or the floor.  This is what happens with liquids.  Very little of the sound beam gets diverted as it goes through the liquid, so the vast majority of the beam hit the structures posterior to the fluid collection.  This is way fluid is so great for ultrasound.  It transmits sound waves to allow us visualization of distal structures, a property called Acoustic Enhancement.




So we have talked about solids, semi-solids, liquids, but what about gases?  Let's go back to the tennis balls.  Imagine if we set up the Bair Hugger hose, shooting air out the end.  Now lets throw out tennis balls through that air current.  What would happen to the tennis balls?


They would go in all directions, right?  We couldn't predict where any of the balls would end up because the air current would disperse them in all directions!  The same is true of ultrasound.  When our sound beam strikes an air interface, the sound beam is scattered in all directions and we cannot see beyond it since the beam is now gone.



The dispersion of sound is what makes us able to hear that fishbowl patient yelling even when we're in Resus.  Although this is an important property for ED management, it is awful for US. 

Well, that's it for today.  I hope you have enjoyed this first ED US Teaching Cast and will take home an important message:  Air is our Enemy and Fluid is our Friend!

Saturday, March 10, 2012

Monday, September 26, 2011

September Case #2: Shortness of Breath

38 y/o AAM presents to the ED with complaints of epigastric pain radiating to his chest for 1 week.  He admits to SOB, but denies N/V/D or cough.  Subjective F/C and generalized body aches.  Recent weight loss of >50 lbs in the past several months.
PMHx:  HTN
PSHx: none
SocHx:  h/o tobaco use, but quit recently; occasional ETOH; marijuana and cocaine use
FamHx; none

PE: BP 173/117, P 101, R 18, T 98.3, Pox 100% (RA)
GEN: WNWD, AAM, NAD
CHEST: CTA B, nonlabored
CV: RRR, IV/VI SEM, +bruit/thrill
ABD:  soft, NTND, +BS
EXT:  no C/C/E, no splinter hemorrhages

Bedside US is performed as the patient is without history of a murmur:



What is your diagnosis?

What is on your differential for etiology of this diagnosis?

Friday, September 9, 2011

September Case #1: Blindness- Discussion

This patient has a normal retina and optic nerve, but appears to have a lens dislocation in the L eye along with some hyperechoic debris in the posterior chamber which may represent vitreous hemorrhage vs scar tissue.

Lens Dislocation

Lens dislocation or ectopia lentis, is a rare condition characterized by a displacement of the lens of the eye.  If the lens lies completely outside of the lens patellar fossa, then it is dislocated or luxated.  Lens subluxation is definied as a partially displaced lens which stil lies in the lens space.  The dislocation of the lens results from the disruption of the zonular fibers of the lens and the degree of disruption is proportional to the degree of lens displacement.

The most common cause of lens dislocation is trauma, but it can also be congential.  It is also associated with Marfan's, homocystinuria, Weil-Marchesani, sulfite oxidase deficiency, and hyperlysinemia.


Patients with lens dislocations usually complain of varying degrees of decreased visual accuity depending if it is a partial or complete dislocation.  In trauma patients, be aware that other ocular signs of trauma may also be present such as retinal detachment, vitreous hemorrhage or globe rupture.

On Ultrasound, the lens is normally located posterior to the iris and anterior chamber and appears as a hyperechoic or white line.

When it is dislocated, the lens will appear as an ovoid structure floating in the posterior chamber.
Be sure to evaluate the eye with the patient looking straight ahead first, then with the eyes looking medial and lateral.  This allows visualization of the entire eye.

Tuesday, September 6, 2011

September Case #1: Blindness

60 y/o AAM presents to the ED with complaints that he is having difficulty seeing out of his R eye over the past several days.  He states that he has not been able to see out of his left eye for about 10 years and never had this complaint evaluated.  He denies pain, trauma, drainage or headache.
PMHx: HTN
PSHx: none
FamHx: DM, CAD
SocHx: +tob (ippd); no ETOH or IVDU

PE: BP 170/83, P 99, R 16, T 98.6, POx 99% (RA)
GEN:  WNWD AAM, NAD
EYE:  visual accuity 20/100 OD, 20/200 OS;  EOMI;  lids, lashes and lacrimals normal;  no conjunctival injection;  no fluoresceine uptake or dendritic lesions;  no FB or rust ring noted;  anterior chamber flat and narrow, no cell and flare; L pupil irregular and minimally reactive;  IOP 16OD, 20OS;  no seidel's sign;  fundoscopic exam limited

Bedside US performed:




What is the diagnosis?